Statistical data collected by various research bodies indicate a close epidemiological relationship between obesity and diabetes. The studies recommend weight cut and increased physical activity as prerequisites for prevention and reduction of the risk of type 2diabetes. Weight loss and exercise are suggested as remedies against contraction of diabetes due to the fact the duo have the potential of decreasing resistance of insulin which is a major physiological defect that is associated with the development or elimination of diabetes. Poor diet and physical activity are the contributing factors to the development of obesity (Astrup, 2010). Studies that have been conducted have proven that once one has been diagnosed with obesity, vulnerability to diabetes remains extremely high.
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Various studies have established that prevalence of childhood overweight expose children to prevalence of diabetes. Thus the risk of diabetes among patients diagnosed with obesity remains high across all the populations and age groups. The age range associated with high risk of diabetes is the aged between 25 and 34 year (Astrup, 2010). Coincidentally, this is also the stage during which most people especially women are diagnosed with obesity due to poor dietary and physical exercise. Studies have thus safely concluded that obesity often leads to diabetes across the demographic categories. Recent studies with designs that included measurement of physical activity, amount of energy spent on different activities and the eating habit of the participants have been conclusive on the between diabetes and obesity.
Prevalence of obesity has been the concern of most health research institutions. Most studies continue to show consistent findings on the relationship between obesity and diabetes across all the age groups (ProQuest, 2012). In the European countries, diabetes has been found to be one of the fastest growing diseases. Currently the disease affects 10-40% adults. The main contributing factor to this accelerating risk of diabetic infections is obesity. According to the studies that have been conducted, there is reliable data that consistently provide the proof that serious co-morbidity like type 2 diabetes and other cardiovascular infections attributable to the spread of obesity. Findings of studies by different epidemiological research organizations indicate that a significant 80-95% of the patients diagnosed with diabetes were obese (Astrup, 2010). Such diabetic infections are largely attributed to overweight and abdominal fat accumulation.
All signs are that diabetic infections are likely to increase given the fact that obesity diagnoses across the age divide are alarmingly a reality. In the developed countries for example, epidemic research bodies have researched and proven that diabetes will increase by almost 27% especially among the adult population (Wee et al, 2012). The explanation for this unfortunate likelihood is the lack of physical exercise, reduced activity and poor diet. This proves that the rise in diabetic infections is significantly attributable to obesity in most of the age groups across the globe. Cross-sectional studies have continued to prove some reliable consistency that high fat, low carbohydrate dietary provision contributes to the development of obesity which in turn promotes the risk of infection with diabetes (Wing et al, 2001).
In order to firm up the claim that obesity is a risk factor in diabetes infection; researchers from the University of Pennsylvania have linked diabetic infections resulting from obesity to a hormone (Maugh, 2001). According to the scientists, a hormone known as resistin is released by the fat cells. This in turn interferes with the activities of insulin leading to the development of diabetes 2. Through this discovery, scientists are now able to clearly account for the scientific relationship between obesity and diabetes. Further, the discovery may lead to the development of drugs that will counter the effects of the hormone that cause insulin resistance. Although this relationship has been established, studies are yet to empirically understand just how the extra pounds from the obese patients often interfere with metabolism of sugar content in the blood stream (Maugh, 2001).
There are studies that nonetheless have not been conclusive. The studies show that although obesity was a predictor of infection with diabetes, adults that are diagnosed with obesity were less likely to be diagnosed with diabetes when compared to adults who did not have obesity. The study by epidemiological researchers from Pennsylvanian University however confirmed that a significant majority of approximately 57% of patients diagnosed with obesity were more vulnerable to diabetic infections (Maugh, 2001).
In conclusion, most of the study findings positively confirm the research question that pointed to the existing relationship between obesity and diabetes. Although some studies still search for the empirical evidence for such relationships, the published findings of most of the epidemic research bodies have been able to prove the relationship scientifically by explaining the role of the hormone resistin. Thus addressing the research question aids and provokes scientific search for new knowledge on the relationship between obesity and diabetes that has led to the discovery of several drugs that thwart development of obesity to diabetes through the growth of the hormone resistin that limits the development of insulin.
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