Type I diabetes is commonly referred to as diabetes mellitus in the medical field. It involves the destruction of the beta cells in the pancreas by the body immune system that synthesizes and secretes insulin to the blood system. Consequently, the body fails to homeostasisally regulate the blood sugar levels (Allman, 2008). This results to a wide range of physiological, biochemical, molecular and heredity malfunctioning in the body of a diabetic invalid person.
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Instigation of type I diabetes has been postulated using various medical postulates. Among the known and postulated molecular, biochemical and physiological basis that lead to destruction of the beta cells include hereditary, toxicological, dietary and physiological precipitous causes. It's from these precipitous causes that trigger destruction of bête cells that prevention and treatment of diabetes should be based. Prevention involves medical research to indentify the dietary, environmental, lifestyle and microbial root causative agent for diabetes mellitus.
Toxicological destruction of beta cells can be avoided through measures to prevent environmental pollution with Type I diabetes, toxicants. Certified toxicological causative agents include pesticides such as pyrinuron. As a measure to prevent this farm and household pesticide toxicological implication, this chemical should be banned in the consumer market. Other convectional, weight manipulating pills that interfere with the intestinal anatomy and its permeability should also be banned in the market. Weight loss or gain therapy should be instigated under the directives of a health physician, and strictly for pathological reasons.
Environmental prevention of diabetes mellitus causation involves maintenance of proper hygiene and proper lifestyles. People living in slums have shown high susceptibility to diabetes I disease. This is attributed to presence of microbial, diabetes causative agents, in the dirty environment such as cocksackie virus, known to be an impetus in beta cell destruction by the immune system. Such a trend calls for government and personal effort to boost the public health conditions in the slums (Watkins, 2003). Having a clean world is not about initiating a mega billion project, if everyone sweeps his courtyard, the whole world will be clean.
Dietary causation of diabetes mellitus can be prevented through proper dietary intake and avoiding foods established as diabetes instigators. Breast feeding a baby for a proper time before weaning, has mildly been certified to prevent diabetes prevalence in the adult phase of the child. Cow milk has also been mildly censured as an autoantibody diabetes triggering agent. This certification doesn't censure cow milk intake. However, it calls for reduced intake in adults and discourages mothers from substituting breast feeding with bottled milk. Wheat has also been mildly censured as a diabetes mellitus triggering agent, and the reason for brown bread intake recommendation as opposed to the white bread. Food recommended for diabetes mellitus prevention includes vitamin D administered in the very first year of infancy (Brown, 2008). Reduction of diabetes mellitus in children immunized with these dietary drugs in subsequent adult life has been calibrated to cases of up to 80%.
Alcoholism and smoking as contributors to pancreases anatomical deformities and malfunctioning are also highly discouraged in diabetes mellitus prevention therapy. Alcoholic destruction of the liver associated with cirrhosis is an impetus to the subsequent malfunctioning of the pancreas, as the two organs are simulative in their functions. This thus calls for reduced alcohol intake and complete no alcohol intake in diabetic. Smoking is a certified carcinogenic agent. Its cause of pancreatic cancer and subsequent treatment therapy are major diabetes mellitus causative agent in destroying islets cells of the pancreas.
Immunological prevention of diabetes mellitus, involves an attempt to biochemically alter the biochemical immunological pathway, that culminate in the destruction of beta cells in the pancreas. Chemicals manufactured for this immunological alteration include cyclosporine A an established immunological inhibitor.
Treatment of Type I diabetes, involves either administration of insulin to the blood stream of a diabetic person, or transplant of pancreas or the islets cells that secrets insulin to the blood stream. Insulin is administered to the bloodstream either through injections, insulin pump or inhalation (Tattarsals, 2009). However, this insulin administration is strictly monitored to avoid complication resulting from excess insulin in the blood stream above the optimal requirement. Diabetic persons are also given dietary recommendation as an attempt to manage their blood sugar levels. Carbohydrates foods are the majorly censured food in diabetic therapy. Lifestyle, pathological aggravators such as smoking is also censured as a management attempt to contain this disease.
Pancreas and islet's cells transplant are only conducted in severe cases of diabetes. It involves a precursor donor and recipient analysis of the organs compatibility to avoid rejection reprisals.
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